有哪些信誉好的足球投注网站alms1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosisalms1-deficient成纤维细胞过表达的细胞外基质成分,显示细胞周期延迟和抗细胞凋亡.pdfVIP
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alms1-deficient fibroblasts over-express extra-cellular matrix components, display cell cycle delay and are resistant to apoptosisalms1-deficient成纤维细胞过表达的细胞外基质成分,显示细胞周期延迟和抗细胞凋亡
ALMS1-Deficient Fibroblasts Over-Express Extra-Cellular
Matrix Components, Display Cell Cycle Delay and Are
Resistant to Apoptosis
1. 1. 1 2 3
Elisabetta Zulato , Francesca Favaretto , Caterina Veronese , Stefano Campanaro , Jan D. Marshall , Sara
1 4 3 5 6 7 ¨
Romano , Anna Cabrelle , Gayle B. Collin , Barbara Zavan , Anna S. Belloni , Enrica Rampazzo , Jurgen K.
3 5 1 1 1 1
Naggert , Giovanni Abatangelo , Nicola Sicolo , Pietro Maffei , Gabriella Milan *, Roberto Vettor
1 Department of Medical and Surgical Sciences, University of Padua, Padua, Italy, 2 Department of Biology, CRIBI (Centro Ricerca Interdipartimentale Biotecnologie
Innovative), University of Padua, Padua, Italy, 3 The Jackson Laboratory, Bar Harbor, Maine, United State of America, 4 Department of Clinical and Experimental Medicine,
University of Padua, Padua, Italy, 5 Department of Histology, Microbiology and Biomedical Technologies, University of Padua, Padua, Italy, 6 Department of Human
Anatomy and Physiology, University of Padua, Padua, Italy, 7 Department of Oncology and Surgical Sciences, University of Padua, Padua, Italy
Abstract
¨
Alstrom Syndrome (ALMS) is a rare genetic disorder (483 living cases), characterized by many clinical manifestations,
including blindness, obesity, type 2 diabetes and cardiomyopathy. ALMS is caused by mutations in the ALMS1 gene,
encoding for a large prot
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