有哪些信誉好的足球投注网站comparative analysis of mrna isoform expression in cardiac hypertrophy and development reveals multiple post-transcriptional regulatory modules比较分析的mrna表达同种型心脏肥大和发展揭示了多个转录后调节模块.pdfVIP
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comparative analysis of mrna isoform expression in cardiac hypertrophy and development reveals multiple post-transcriptional regulatory modules比较分析的mrna表达同种型心脏肥大和发展揭示了多个转录后调节模块
Comparative Analysis of mRNA Isoform Expression in Cardiac Hypertrophy and Development Reveals Multiple Post-Transcriptional Regulatory Modules 1,3 2 2 1 2 1 Ji Yeon Park , Wencheng Li , Dinghai Zheng , Peiyong Zhai , Yun Zhao , Takahisa Matsuda , 1 1 2,3 Stephen F. Vatner , Junichi Sadoshima , Bin Tian * 1 Department of Cell Biology and Molecular Medicine, University of Medicine and Dentistry of New Jersey, Newark, New Jersey, United States of America, 2 Department of Biochemistry and Molecular Biology, University of Medicine and Dentistry of New Jersey, Newark, New Jersey, United States of America, 3 New Jersey Medical School and Graduate School of Biomedical Science, University of Medicine and Dentistry of New Jersey, Newark, New Jersey, United States of America Abstract Cardiac hypertrophy is enlargement of the heart in response to physiological or pathological stimuli, chiefly involving growth of myocytes in size rather than in number. Previous studies have shown that the expression pattern of a group of genes in hypertrophied heart induced by pressure overload resembles that at the embryonic stage of heart development, a phenomenon known as activation of the ‘‘fetal gene program’’. Here, using a genome-wide approach we systematically defined genes and pathways regulated in short- and long-term cardiac hypertrophy conditions using mice with transverse aortic constriction (TAC), and compared them with those regulated at different stages of embryonic and postnatal development. In addition, exon-level analysis revealed widespread mRNA isoform changes during cardiac hypertrophy resulting fro
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