有哪些信誉好的足球投注网站anti-il-2 treatment impairs the expansion of treg cell population during acute malaria and enhances the th1 cell response at the chronic diseaseanti-il-2损害treg细胞群的扩张治疗在急性疟疾和提高th1细胞反应的慢性疾病.pdfVIP
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anti-il-2 treatment impairs the expansion of treg cell population during acute malaria and enhances the th1 cell response at the chronic diseaseanti-il-2损害treg细胞群的扩张治疗在急性疟疾和提高th1细胞反应的慢性疾病
Anti-IL-2 Treatment Impairs the Expansion of Treg Cell
Population during Acute Malaria and Enhances the Th1
Cell Response at the Chronic Disease
´ 1 1,2 3 1
Claudia A. Zago *, Karina R. Bortoluci , Luiz R. Sardinha , Fernando D. Pretel , Sheyla I. Castillo-
´ 1 ´ 1 1 1 ´
Mendez , Ana Paula Freitas do Rosario , Meire I. Hiyane , Sandra M. Muxel , Sergio M. Rodriguez-
´ 1 1 ´ ´ 1 ´ 1
Malaga , Ises A. Abrahamsohn , Jose M. Alvarez , Maria Regina D’Imperio Lima
ˆ ´ ˜ ˜ ˜ ˆ ´
1 Departamento de Imunologia, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Sao Paulo, Brasil, 2 Departamento de Ciencias Biologicas (Campus
˜ ˜ ˜
Diadema) and Centro de Terapia Celular e Molecular (CTC-Mol), Universidade Federal de Sao Paulo, Sao Paulo, Sao Paulo, Brasil, 3 Instituto Israelita de Ensino e Pesquisa
˜ ˜
Albert Einstein, Sao Paulo, Sao Paulo, Brasil
Abstract
Plasmodium chabaudi infection induces a rapid and intense splenic CD4+ T cell response that contributes to both disease
pathogenesis and the control of acute parasitemia. The subsequent development of clinical immunity to disease occurs
concomitantly with the persistence of low levels of chronic parasitemia. The suppressive activity
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