第三节腐蚀性食管灼伤(Third corrosive esophageal burns).docVIP

第三节腐蚀性食管灼伤(Third corrosive esophageal burns).doc

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第三节腐蚀性食管灼伤(Third corrosive esophageal burns)

第三节腐蚀性食管灼伤(Third corrosive esophageal burns) Third corrosive esophageal burns Corrosive esophageal burns (erosive, burn, of, esophagus) are mostly caused by swallowing acid or strong alkali and other chemical corrosive agents Chemical burn of esophagus. There are also long-term reflux esophagitis, long-term consumption of vinegar or long-term use of acidic drugs (such as strength) A chemical burn of the esophagus caused by a bacterium, tetracycline, aspirin, etc., but rare. Strong bases produce more severe dissolution Necrotic necrosis; a strong acid producing protein; coagulation necrosis. The pathological severity of esophageal chemical burns, type, concentration, dosage, decided to swallow esophageal chemical corrosion agent Anatomical features, concomitant vomiting, and time of contact between the etchant and the tissue. Swallow chemical corrosion, burn site often is not limited to the esophagus, often including oropharynx, larynx, stomach or twelve fingers Intestine. Usually, the corrosive agent has the longest contact with the three physiological segments of the esophagus, so it usually occurs more widely in these sites Burn. According to the pathological degree of burn, it can be divided into I degree, degree and FFF degree. The degree of burn is: I degree: the esophagus mucosa is superficial Blood edema, after the desquamation period, 7-8 days after the recovery, leaving no paralysis marks. Oll degree: burns involving the muscularis of the esophagus. stay Acute phase of tissue congestion, edema, exudation, tissue necrosis, shedding, ulceration. Granulation tissue hyperplasia occurred within 3-6 weeks. Later, the fibrous tissue formed a scar and narrowed. L degree: coagulation and necrosis of the whole layer of esophagus and its surrounding tissues can lead to food Perforation of tube and mediastinum. The pathological process of burns can be divided into three stages. The first stage is inflammation and edema in the first few days after injury Or necrosis. Sy

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