有哪些信誉好的足球投注网站helicobacter pylori colonization ameliorates glucose homeostasis in mice through a ppar γ-dependent mechanism幽门螺杆菌的殖民改善小鼠的葡萄糖稳态通过pparγ-dependent机制.pdfVIP
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helicobacter pylori colonization ameliorates glucose homeostasis in mice through a ppar γ-dependent mechanism幽门螺杆菌的殖民改善小鼠的葡萄糖稳态通过pparγ-dependent机制
Helicobacter pylori Colonization Ameliorates Glucose Homeostasis in Mice through a PPAR c-Dependent Mechanism 1,4 2 1 1 Josep Bassaganya-Riera *, Maria Gloria Dominguez-Bello , Barbara Kronsteiner , Adria Carbo , 1 1 1 1 1¤ Pinyi Lu , Monica Viladomiu , Mireia Pedragosa , Xiaoying Zhang , Bruno W. Sobral , 1 1 1 1 3 Shrinivasrao P. Mane , Saroj K. Mohapatra , William T. Horne , Amir J. Guri , Michael Groeschl , Gabriela Lopez-Velasco1, Raquel Hontecillas1 1 Nutritional Immunology and Molecular Medicine Laboratory, Center for Modeling Immunity to Enteric Pathogens, Virginia Bioinformatics Institute, Virginia Tech, ´ Blacksburg, Virginia, United States of America, 2 Department of Biology, University of Puerto Rico, Rıo Piedras. San Juan, Puerto Rico, United States of America, 3 Erlangen University, Erlangen, Germany, 4 Department of Biomedical Sciences and Pathobiology, VA-MD Regional College of Veterinary Medicine, Virginia Tech, Blacksburg, Virginia, United States of America Abstract Background: There is an inverse secular trend between the incidence of obesity and gastric colonization with Helicobacter pylori, a bacterium that can affect the secretion of gastric hormones that relate to energy homeostasis. H. pylori strains that carry the cag pathogenicity island (PAI) interact more intimately with
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