西安交通大学医学院呼吸系统疾病课件 pulmonary tuberculosis.pptVIP

B. Groups (Pools) Group A metabolically active early focus,cavity Group B relatively metabolically inactive Group C occasionally metabolism caseous necrosis Group D ‘dormant group INH, RFP, SM PZA RFP RFP 5mm (–) 5~9mm (+) 10~19mm (++) 20mm (+++) with blister,ulcer Hematogenous disseminated P.TB Contamination and development primary TBforcus acute Subcute chronic resistance Hematogenous disseminated P.TB Acute Size, distribution, density uniform 2mm Subacute Size, distribution, density different chronic Contamination and development \ Contamination and development infiltrative pulmonary tuberculosis TB left by primary infection immunity proliferation Infiltrative TB TB allergy Cheesy necrosis cavity spread along Bs tuberculoma Contamination and development infiltrative pulmonary tuberculosis most common Frequent in adult Slow onset Symptom depend on focus and reactivity Predilection site: upper or lower of clavicle Contamination and development Contamination and development chronic fibro-cavernous pulmonary tuberculosis Long history Recurrent symptom Smear-TB+ Contamination and development chronic fibro-cavernous pulmonary tuberculosis thick walled Cavity Upward retraction of hilar marking Spread lesion along bronchus Pleura thickening weeping willowed lung marking mediastinum moved compensatory emphysema Fribrosis—volume loss Contamination and development Type IV —tuberculous pleurisy Contamination and development Symptoms tuberculosis toxic symptoms weight loss low-grade fever night sweats, no appetite malaise Clinical manifestation Symptoms respiratory symptoms usually insidious Cough white sputum Hemoptysis chest pain Severe dyspnea Clinical manifestation Sign Small lesion or in deep place abscent Large area consolidation Rales at the clavicles or interscapular region Extensive