cxcr6, a newly defined biomarker of tissue-specific stem cell asymmetric self-renewal, identifies more aggressive human melanoma cancer stem cellscxcr6,新定义的生物标志物的组织干细胞自我更新不对称,标识更激进的人类黑色素瘤肿瘤干细胞.pdfVIP

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cxcr6, a newly defined biomarker of tissue-specific stem cell asymmetric self-renewal, identifies more aggressive human melanoma cancer stem cellscxcr6,新定义的生物标志物的组织干细胞自我更新不对称,标识更激进的人类黑色素瘤肿瘤干细胞.pdf

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cxcr6, a newly defined biomarker of tissue-specific stem cell asymmetric self-renewal, identifies more aggressive human melanoma cancer stem cellscxcr6,新定义的生物标志物的组织干细胞自我更新不对称,标识更激进的人类黑色素瘤肿瘤干细胞

CXCR6, a Newly Defined Biomarker of Tissue-Specific Stem Cell Asymmetric Self-Renewal, Identifies More Aggressive Human Melanoma Cancer Stem Cells 1 2 1 3 4 4 Rouzbeh Taghizadeh , Minsoo Noh , Yang Hoon Huh , Emilio Ciusani , Luca Sigalotti , Michele Maio , 5 6 7 1. 8 . Beatrice Arosio , Maria R. Nicotra , PierGiorgio Natali , James L. Sherley , Caterina A. M. La Porta * 1 Programs in Regenerative Biology and Cancer Biology, Adult Stem Cell Technology Center, Boston Biomedical Research Institute, Watertown, Massachusetts, United States of America, 2 College of Pharmacy, Ajou University, Suwon, Republic of Korea, 3 Istituto Neurologico Carlo Besta, Milano, Italy, 4 Cancer Bioimmunotherapy Unit, Department of Medical Oncology, Centro di Riferimento Oncologico, Istituto di Ricovero e Cura a Carattere Scientifico, Aviano, Italy, 5 Department of Internal Medicine, ` Universita degli Studi di Milano, Milano, Italy, 6 Molecular Biology and Pathology Institute, National Research Council, Roma, Italy, 7 CINBO Laboratory, University Chieti, Chieti, Italy, 8 Department of Biomolecular Science and Biotechnology, University of Milan, Milan, Italy Abstract Background: A fundamental problem in cancer research is identifying the cell type that is capable of sustaining neoplastic growth and its origin from normal tissue cells. Recent investigations of a variety of tumor types have shown that phenotypically identifiable and isolable subfractions of cells possess the tumor-forming ability. In the present paper, using two lineage-related human melanoma cell