有哪些信誉好的足球投注网站complex exon-intron marking by histone modifications is not determined solely by nucleosome distribution复杂exon-intron由组蛋白标记修改不完全由核小体分布.pdfVIP
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complex exon-intron marking by histone modifications is not determined solely by nucleosome distribution复杂exon-intron由组蛋白标记修改不完全由核小体分布
Complex Exon-Intron Marking by Histone Modifications Is Not Determined Solely by Nucleosome Distribution 1.¤a 2. 1.¤b 1.¤c 2 Pawandeep Dhami , Peter Saffrey , Alexander W. Bruce , Shane C. Dillon , Kelly Chiang , 2 1 1 1 3 1 Nicolas Bonhoure , Christoph M. Koch , Jackie Bye , Keith James , Nicola S. Foad , Peter Ellis , 3 1,3 1 1 1¤d Nicholas A. Watkins , Willem H. Ouwehand , Cordelia Langford , Robert M. Andrews , Ian Dunham , David Vetrie1,2* 1The Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, United Kingdom, 2 Institute of Cancer Sciences, University of Glasgow, United Kingdom, 3 Department of Haematology, University of Cambridge and NHS Blood and Transplant Cambridge, Cambridge, United Kingdom Abstract It has recently been shown that nucleosome distribution, histone modifications and RNA polymerase II (Pol II) occupancy show preferential association with exons (‘‘exon-intron marking’’), linking chromatin structure and function to co- transcriptional splicing in a variety of eukaryotes. Previous ChIP-sequencing studies suggested that these marking patterns reflect the nucleosomal landscape. By analyzing ChIP-chip datasets across the human genome in three cell types, we have found that this marking system is far more complex than previously observed. We show here that a rang
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