肝脂肪变和脂肪肝的病理变化（Pathological changes of hepatic steatosis and fatty liver）.docVIP

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肝脂肪变和脂肪肝的病理变化（Pathological changes of hepatic steatosis and fatty liver）.doc

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肝脂肪变和脂肪肝的病理变化（Pathological changes of hepatic steatosis and fatty liver）

肝脂肪变和脂肪肝的病理变化（Pathological changes of hepatic steatosis and fatty liver） Pathological changes of hepatic steatosis and fatty liver Shandong Medicine 2000 thirteenth, fortieth volumes Author: Zhang Tingguo Setting: Shandong Medical University, 250021 The lipid droplets in the cytoplasm are more than the physiological range, or fat droplets are present in normal cells without lipid droplets, which is called fat change. The liver is the main organ of fatty acid metabolism, liver fatty change is more common, when serious, almost all liver cells have fat change, liver swelling, can have mild tenderness and abnormal liver function, called fatty liver. The liver cell can absorb fatty acids and esterified them by blood, and can also synthesize fatty acids from carbohydrates. This absorption or newly synthesized fatty acid is only a small part of liver cells as energy use; most of it in the form of protein and ester combination before the formation of lipoprotein, blood, and then stored in the fat library, or for other organizations to use; there is a small part of the phospholipids and other lipids combined with protein, carbohydrate, forming the structure components of cells, which become fat. Therefore, any obstacle to the process may lead to fatty degeneration of the liver cells. The mechanism of hepatic steatosis has the following aspects: (1) the obstruction of lipoprotein synthesis can not be transported out of fat, so it results in the accumulation of fat in liver cells. Due to raw materials (such as synthetic lipoprotein phospholipids or phospholipid composition of choline and other substances) or lack of toxic chemicals (such as alcohol, carbon tetrachloride) and other toxins (such as mycotoxins) destruction of endoplasmic reticulum, or inhibit the activity of some enzymes, the composition of lipoprotein and lipoprotein phospholipid and protein synthesis obstacles caused by. Neutral fat synthesis is too much. Because of hunger or certain diseases (such as digest