有哪些信誉好的足球投注网站mutations in complement regulatory proteins predispose to preeclampsia a genetic analysis of the promisse cohort补体调节蛋白的突变引起子痫前期的遗传分析promisse队列.pdfVIP
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mutations in complement regulatory proteins predispose to preeclampsia a genetic analysis of the promisse cohort补体调节蛋白的突变引起子痫前期的遗传分析promisse队列
Mutations in Complement Regulatory Proteins Predispose to Preeclampsia: A Genetic Analysis of the PROMISSE Cohort 1 2 3 3 4 Jane E. Salmon *, Cara Heuser , Michael Triebwasser , M. Kathryn Liszewski , David Kavanagh , Lubka 5 2 4 5 3 Roumenina , D. Ware Branch , Tim Goodship , Veronique Fremeaux-Bacchi , John P. Atkinson 1 Autoimmunity and Inflammation Program, Hospital for Special Surgery, Cornell Weill Medical College, New York, New York, United States of America, 2 Department of Obstetrics and Gynecology, University of Utah, Salt Lake City, Utah, United States of America, 3 Department of Medicine/Division of Rheumatology, Washington University School of Medicine, St. Louis, Missouri, United States of America, 4 Institute of Human Genetics, Newcastle University, Newcastle upon Tyne, United Kingdom, 5 Assistance ˆ ´ Publique-Hopitaux de Paris, Hopital Europeen Georges-Pompidou, Service d’Immunologie Biologique, Paris, France Abstract Background: Pregnancy in women with systemic lupus erythematosus (SLE) or antiphospholipid antibodies (APL Ab)— autoimmune conditions characterized by complement-mediated injury—is associated with increased risk of preeclampsia and miscarriage. Our previous studies in mice indicate that complement activation targeted to the placenta drives angiogenic imbalance and placental insufficiency. Methods and Findings: We use PROMISSE, a prospective study of 250 pregnant patients with SLE and/or APL Ab, to test the hypothesis in humans that impaired capacity to limit complement activation predisp
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