有哪些信誉好的足球投注网站a shared role for rbf1 and dcap-d3 in the regulation of transcription with consequences for innate immunity共享角色rbf1和dcap-d3对先天免疫转录调节的影响.pdfVIP
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a shared role for rbf1 and dcap-d3 in the regulation of transcription with consequences for innate immunity共享角色rbf1和dcap-d3对先天免疫转录调节的影响
A Shared Role for RBF1 and dCAP-D3 in the Regulation of Transcription with Consequences for Innate Immunity 1 2,3 2 4 5 Michelle S. Longworth *, James A. Walker , Endre Anderssen , Nam-Sung Moon , Andrew Gladden , 6 2 2 Margarete M. S. Heck , Sridhar Ramaswamy , Nicholas J. Dyson 1 Department of Molecular Genetics, The Lerner Research Institute, Cleveland Clinic, Cleveland, Ohio, United States of America, 2 Massachusetts General Hospital Cancer Center and Harvard Medical School, Charlestown, Massachusetts, United States of America, 3 Center for Human Genetic Research, Massachusetts General Hospital, Boston, Massachusetts, United States of America, 4 Department of Biology, Developmental Biology Research Initiative, McGill University, Montreal, Canada, 5 Department of Genetics, The University of Texas MD Anderson Cancer Center, Houston, Texas, United States of America, 6 Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom Abstract Previously, we discovered a conserved interaction between RB proteins and the Condensin II protein CAP-D3 that is important for ensuring uniform chromatin condensation during mitotic prophase. The Drosophila melanogaster homologs RBF1 and dCAP-D3 co-localize on non-dividing polytene chromatin, suggesting the existence of a shared, non-mitotic role for these two proteins. Here, we show that the absence of RBF1 and dCAP-D3 alters the expression of many of the same genes in larvae and adult flies. Strikingly, most of the genes affected by the loss of RBF1 and dCAP-D3 are not classic cell cycle genes but are develop
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