主动脉反搏.pptVIP

130 BPM 60 BPM Re-synchronized Re-synchronized Reference Appendix A and B for Test Conditions P/N 0002-08-0338: S98 Rhythm Comparisons S98XT (Conventional Timing): (Conventional Timing): 自动辨认心率改变 * * Pathophysiology of Cardiogenic shock due to acute myocardial infarction: The pathophysiology of Cardiogenic Shock involves a downward spiral: ischemia causes myocardial dysfunction, which in turn, worsens ischemia. Left ventricular [LV] dysfunction leads to increased sympathetic tone and activation of the renin-angiotensin system [RAS] causing vasoconstriction and sodium retention, two factors which promote further LV dysfunction. Patients in shock often have obstruction of a major coronary vessel, with resultant extensive loss of contractile mass, which leads to decreased arterial pressure. A significant drop in arterial pressure may be profound enough to cause a decrease in coronary blood flow, aggravating myocardial ischemia and further compromising LV function. Initially, the dysfunction is reversible but persistent hypotension leads to irreversible permanent cellular injury and necrosis, organ dysfunction, and can ultimately lead to death. * * * When used earlier in the intervention process, the clinical and economic benefits of Counterpulsation therapy include: proven reduction in mortality, improved patient outcomes, shorter lengths of stay and reduced costs. * * Recommendations during pressure monitoring through the central lumen: Before Pumping: 1. Use a standard flushing apparatus. Careful technique should be used in the set up and flushing of the arterial pressure monitoring apparatus to minimize the risk of an embolus. 2. Minimize the length of pressure tubing. 3. Use only high stiffness pressure tubing [as provided by Datascope or Abbott] 4. Aspirate and discard 3cc of blood from the central lumen prior to attaching pressure tubing. 5 Immediately manually flush with 3-5 cc of flush solution prior t