有关兔的生理学实验(呼吸,血压,尿量)的分析(An analysis of physiological tests (respiration, blood pressure, urine volume) in rabbits).docVIP

有关兔的生理学实验(呼吸,血压,尿量)的分析(An analysis of physiological tests (respiration, blood pressure, urine volume) in rabbits).doc

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有关兔的生理学实验(呼吸,血压,尿量)的分析(An analysis of physiological tests (respiration, blood pressure, urine volume) in rabbits)

有关兔的生理学实验(呼吸,血压,尿量)的分析(An analysis of physiological tests (respiration, blood pressure, urine volume) in rabbits) Increase the invalid cavity, inhalation of CO2, lactate injection, occlusion of the right carotid artery, injection of glucose, depressor nerve stimulation, vagus nerve, injection of norepinephrine (NE) and acetylcholine (ACh), furosemide, observed and recorded changes of arterial blood pressure, respiration, urine volume 1. increase the invalid increase of rabbit airway length after blood pressure compared with the control group significantly increased the experimental cavity effect on circulation and respiration in urinary and respiratory (P0.01), (P0.01), to speed up the increased urine volume compared with the control group, no significant difference (P0.05). The increase of airway length equal to the increase void cavity, increase the update rate of alveolar gas void cavity caused by blood decreased, PCO2 increased, PO2 decreased, hypoxia and carbon dioxide can stimulate increased aortic body and carotid chemoreceptor, excited nerve respectively through the aortic sinus nerve afferent and respiratory and vasomotor center, a cause the respiratory excitement, respiration increased; on the other hand caused vasoconstriction excitability, increased peripheral vascular tension. In addition, the increase of carbon dioxide in the blood can also directly stimulate the medulla oblongata vascular center. From the afferent chemoreceptor impulse on medullary heart inhibition of central excitatory effects, such as in artificially pulmonary ventilation under the condition of same organ stimulation of carotid body chemistry, can cause cardiac slowing, myocardial contractility decreased, myocardial oxygen consumption and reduced cardiac output. In natural respiration, however, when the chemoreceptor stimulation accelerates respiration, the pulmonary stretch reflex can subsequently cause the heart rate to increase without the inhibition of cardiac activity. Thus, fr

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