双腔起搏高级功能.ppt

* * * * * * * * * * * Affinity, Integrity, Identity, ADx and Victory- With AutoCapture OFF, AVD is limited to 350ms * * * * * * * * * * The timers for AICS, Hysteresis, AC threshold tests start in manufacturing, from the time the software is downloaded into the device not programming them on. VIP is the exception; the first 30 extension will occur once programmed On, but will then follow the real time clock * * The 3 consecutive R waves outside a search interval that will activate VUP is not tied to the Cycle Count. It has to be three even if the Cycle Count is 1 of 2. A PVC will not initiate VIP * * * * * * Hypertrophic cardiomyopathy is a disease of heart muscle characterized by inappropriate thickening (hypertorphy) of one or more regions of the myocardium in the absence of systemic hypertension or valvular aortic stenosis. Often the hypertrophy is especially prominent in the basal area of the interventricular septum in the left ventricle, where it bulges into the left ventricular outflow tract below the aortic valve. Patients with HCM with septal hypertorphy may develop a persistent or intermittent functional obstruction to blood flow out of the left ventricle during systole. During systole, bulging of the thickened septum into the left ventricular outflow tract (LVOT) causes a narrowing of the LVOT, restricting the ejection of blood from the left ventricle. The acceleration of blood through the narrowed outflow tract can cause the anterior leaflet of the mitral valve to be pulled into the outflow tract, contacting the bulging septum. This is referred to as Systolic Anterior Motion of the mitral valve leaflet, or SAM, Mid-systolic mitral-septal contact resulting from SAM comletes the obstruction to blood flow out of the left ventricle. It also leads to mitral valvular insufficiency and thus mitral regurgitation. The obstruction to blood flow causes excessive pressure to be built up within the left ventricle, such that during systole the pressure within th