系统性红斑狼疮肺动脉高压的临床观察（Clinical observation of pulmonary arterial hypertension in patients with systemic lupus erythematosus）.docVIP

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系统性红斑狼疮肺动脉高压的临床观察（Clinical observation of pulmonary arterial hypertension in patients with systemic lupus erythematosus）.doc

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系统性红斑狼疮肺动脉高压的临床观察（Clinical observation of pulmonary arterial hypertension in patients with systemic lupus erythematosus）

系统性红斑狼疮肺动脉高压的临床观察（Clinical observation of pulmonary arterial hypertension in patients with systemic lupus erythematosus） Clinical observation of pulmonary arterial hypertension in patients with systemic lupus erythematosus 2011-01-16 Zhang Fengchun, Zhang Xiaoping, Jin Nie, Dong Yi Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by multisystem and multiorgan damage. In the past, the incidence of pulmonary hypertension SLE in patients with PAH was very low, and there was no specific treatment [1], which has not attracted enough attention in china. To this end, we investigated the incidence of SLE in patients with PAH, the correlation between PAH and other clinical manifestations of SLE, autoantibodies, and changes in PAH after treatment to improve our understanding of SLE combined with PAH. 1 data and methods 1.1 case selection A total of 43 patients enrolled in the Department of Rheumatology from Peking Union Medical College Hospital from September 1997 to December were enrolled. All patients met the 1982 ARA revised SLE classification criteria. The disease activity is judged by the SLEDAI standard [2]. There were 5 males and 38 females, aged 14~59 years, mean (33 + 11) years, with a course of disease ranging from 2 months to 13 years, with an average of (46 + 52) months. 1.2 method 1.2.1 and PAH methods: ECG and chest X-ray examination were performed after admission and SLE remission, respectively, UCG (according to three tricuspid regurgitation pressure PAP). The diagnostic standard of 1.2.2 PAH 3, 4 UCG showed resting pulmonary artery systolic pressure (PASP) = second mm 30 Hg or pulmonary valve with at least one of the following sound hyperfunction of ECG showed right ventricular hypertrophy or right atrial enlargement; the chest showed pulmonary artery and its branches increased significantly; the UCG right ventricular overload (right ventricular dilatation and three tricuspid regurgitation). There is no history of rheumatic heart dise