第八章白血病概述（The eighth chapter is leukemia overview）.docVIP

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第八章白血病概述（The eighth chapter is leukemia overview）.doc

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第八章白血病概述（The eighth chapter is leukemia overview）

第八章白血病概述（The eighth chapter is leukemia overview） The eighth chapter is leukemia overview ? This chapter centers 1. leukemia characteristics of 2. type of acute leukemia (1) morphologytyping (2) immunophenotyping (3) cytogenetic typing (4) to observe a characteristics of the 1. concepts of molecular biology, leukemia type 3. acute leukemia is a malignant hematopoietic system, is a group of highly malignant hematological diseases the heterogeneity of the characteristics of leukemia cells showed abnormal hyperplasia with maturation disorder. 2. the clinical features of acute leukemia clinical anemia degree; hemorrhage; fever and liver and spleen; lymph nodes; sternal segment can appear local tenderness, joint and bone pain; eye visible granulocytic sarcoma or chloroma; gingival hyperplasia and swelling; skin blue gray maculopapule; testis appeared painless enlargement, multiple to the side; central nervous system disorders such as headache, dizziness, severe vomiting, stiff neck, and even convulsions, coma. Two 、 acute leukemia typing, 1. cell morphological classification (1) FAB collaborative component typing. (2) classification of our country: revised at the Tianjin conference in 1986, see table 2-16-1. Table 2-16-1 classification criteria of acute leukemia in China Subtype classification standard M0 acute myeloid leukemia cell differentiation, the original is more than 30%, T, B lymphoid markers, at least an expression of myeloid antigen, immunohistochemistry or electron microscopy MPO positive M1 original undifferentiated myeloid leukemia, bone marrow myeloblasts (NEC = 90%), promyelocytic rarely, myelocyte stage rare or no. POX or SB (+) + 3% M2, primary granulocytic leukemia, partial differentiation In M2a, the number of granulocytes in the bone marrow ranged from 3% to 90% (NEC), and the number of monocytes was less than 20%. Early fine grained following stage 10% M2b bone marrow myeloid hyperplasia, abnormal primitive early promyelocytic cells increased si