有哪些信誉好的足球投注网站full restoration of brucella-infected dendritic cell functionality through vγ9vδ2 t helper type 1 crosstalk完全恢复brucella-infected树突状细胞功能通过vγ9vδ2辅助t 1型相声.pdfVIP
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full restoration of brucella-infected dendritic cell functionality through vγ9vδ2 t helper type 1 crosstalk完全恢复brucella-infected树突状细胞功能通过vγ9vδ2辅助t 1型相声
Full Restoration of Brucella-Infected Dendritic Cell Functionality through Vc9Vd2 T Helper Type 1 Crosstalk 1,2,3 1,2,3 4 4 1,2,3 Ming Ni , Delphine Martire , Emmanuel Scotet , Marc Bonneville , Francoise Sanchez , Virginie Lafont1,2,3* ´ ´ ` ´ 1 Universite Montpellier 1, Centre d’etudes d’agents Pathogenes et Biotechnologies pour la Sante (CPBS) Montpellier, France, 2 CNRS, UMR 5236, CPBS, F-34965 ´ ´ ´ Montpellier, France, 3 Universite Montpellier 2, CPBS, F-34095 Montpellier, France, 4 Inserm Unite Mixte de Recherche 892, Centre de Recherche en Cancerologie Nantes- ´ ´ Angers, Institut de Recherche Therapeutique Universite de Nantes, Nantes, France Abstract Vc9Vd2 T cells play an important role in the immune response to infectious agents but the mechanisms contributing to this immune process remain to be better characterized. Following their activation, Vc9Vd2 T cells develop cytotoxic activity against infected cells, secrete large amounts of cytokines and influence the function of other effectors of immunity, notably cells playing a key role in the initiation of the adaptive immune response such as dendritic cells. Brucella infection dramatically impairs dendritic cell maturation and their capacity to present antigens to T cells. Herein, we investigated whether V T cells have the ability to resto
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